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This secondary binding site may underlie some of the peptide's reported cytoprotective and anti-inflammatory properties observed in preclinical models, though the clinical significance remains under investigation

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HNF4-depleted HepG2 cells also metabolically clustered together with SNU449 cells but not with control HepG2 in a cluster analysis of all significantly changed metabolites in HNF4-depleted HepG2 cells (Supplementary Fig

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