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For both therapeutic classes, this increased risk of gallbladder or biliary diseases has been thought to be driven by the inhibition of the secretion of cholecystokinin by GLP-1, leading to impaired gallbladder motility and contractility [64, 65]

Studies were considered to have low heterogeneity with I statistic of 25%-50%, moderate heterogeneity with 50%-75%, and high heterogeneity with greater than 75%