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Similarly, SR-L1 has a role in neuronal cell survival by modulating c-Jun N-terminal kinase (JNK)-mediated apoptosis (172)

Higher mitochondrial metabolism in the neurons result in generation of Complex I dependent hydrogen peroxide and neuronal membranes, rich in polyunsaturated fatty acids, are particularly vulnerable to oxidative stress due to their possession of unsaturated double bonds

GSH from the intestinal lumen plays different important roles such as reduction of dietary disulfides, detoxification of xenobiotics, metabolism of peroxidized lipids and maintenance of the mucus fluidity[29,34,41]

This hypoxic state stabilizes hypoxia-inducible factor (HIF-1), a crucial transcription factor mediating the protective effect of SCFAs