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Microglia, astrocytes, and infiltrating macrophages remain chronically activated and secrete neurotoxic factors, while dysfunctional oligodendrocytes and Schwann cells fail to uphold myelin and metabolic support

In this study, we provide a comprehensive clinical and neuropathologic characterization of an APP V717I mutation carrier, shedding new light on the phenotypic variability observed in ADAD cases (Lloyd et al., 2020)

reported that in MITOL knockdown-mediated ferroptosis in cardiomyocytes, knockdown of ChaC1 reversed cell injury and increased GSH [53]