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On the other hand, fibrosis may contribute to oxidative stress or both of them may be triggered by an independent mechanism

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Because the peptide potently and aggressively stimulates angiogenesis via the VEGF/VEGFR2 pathways, there is a theoretical, but highly biologically plausible, risk that BPC-157 could inadvertently supply massive new blood flow to existing, undiagnosed malignant tumors, thereby acting as a catalyst for rapid cancer metastasis

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