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That fragment acts on a target the same Washington State group helped pin down: the so-called AT4 receptor, which was identified as IRAP, insulin-regulated aminopeptidase, an enzyme rather than a classical receptor (Albiston et al., J Biol Chem 2001)

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Schisandrin B also ameliorated A-induced oxidative stress and neuronal dysfunction in the cortex and protected primary cortical neurons from A142 toxicity by regulating apoptotic pathways, including the upregulation of BCL-2 and the suppression of BAX, cytochrome c release and caspase activation [61, 62]

(PubMed) Yu FF, Sun L, Zhou GY, Ping ZG, Guo X, Ba Y