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Interestingly, PYY 136 binds to Y1R, Y2R, and Y5R, whereas PYY 336 has a high affinity for Y2R [80]

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demonstrated that BMSCs modified with insulin-like growth factor binding protein 3 (IGFBP-3) can suppress the proliferative activity of human PASMCs and induce their apoptosis by activating the PI3K/Akt and Ras-mitogen-activated protein kinase (MAPK) signaling pathways (Cheng et al., 2017a)