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612 NMN treatment improves the neuronal functions, including rescuing the memory and cognition in rodent models for Alzheimers disease, protecting neurons from cell death after intracerebral hemorrhage or ischemia, recovering severe retinal degeneration and restoring the age-associated loss of the neural stem/progenitor pool

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Given that PI3K is an important hub in the function of insulin/IGF-1-dependent regulation, this kinase has been inhibited by PTEN phosphatase (see above) and SH2-containing inositol 5-phosphatase 2 (SHIP2) which is enriched in mouse brain and its overexpression impaired memory formation [53] (Fig